Low testicular volume is often the first physical sign of an underlying fertility disorder. Whether the cause is hormonal deficiency, genetic conditions like Klinefelter Syndrome, or early testicular damage, Dr. Adnan Jabbar in Lahore provides precise orchidometric assessment and targeted therapy to maximize reproductive potential.
Normal adult testicular volume is 15-25 mL. Below 12 mL strongly correlates with impaired spermatogenesis and reduced fertility potential.
Approximately 85% of testicular volume is comprised of seminiferous tubules — the factories where sperm are made. Small testes directly mean less sperm-producing tissue, which is why orchidometry is a critical first step in any male fertility evaluation.
Lack of FSH/LH stimulation prevents normal testicular development. Often seen in Hypogonadotropic Hypogonadism. Treatable with gonadotropins.
Klinefelter Syndrome (47,XXY), Y-chromosome microdeletions, or other chromosomal abnormalities cause inherently small, dysfunctional testes with severly impaired sperm production.
Undescended testes (cryptorchidism), childhood mumps orchitis, testicular torsion, or varicocele-induced atrophy can cause irreversible shrinkage and spermatogenic decline.
Dr. Adnan Jabbar systematically identifies the underlying cause before selecting the optimal fertility pathway.
If low testicular volume is caused by hormonal deficiency (HH), gonadotropin therapy (hCG + hMG) can increase testicular size by 50-100% over 6-18 months, initiating spermatogenesis from dormant tubules.
For men with primary testicular failure and severely small testes, Micro-TESE under high-powered microscopy can identify the rare islands of active spermatogenesis deep within the testicular tissue.
If a large varicocele has caused progressive testicular atrophy, microsurgical varicocelectomy can halt further damage and, in some cases, partially restore testicular volume and sperm production over 6-12 months.
Emerging stem cell therapies are being explored to regenerate damaged seminiferous tubules and restore spermatogenesis in men with severe testicular atrophy — a frontier Dr. Adnan Jabbar is actively pioneering.
Small testes are not merely a cosmetic concern — they are a clinical biomarker of reduced sperm-producing tissue. Dr. Adnan Jabbar is one of Pakistan's foremost experts in male reproductive assessment, utilizing precise orchidometry, hormonal profiling, karyotyping, and advanced scrotal ultrasound to determine the exact cause and design the most effective fertility recovery strategy.
Understanding the link between testicular size and fertility.
A normal adult testicular volume ranges from 15-25 mL per testis. Volumes below 12 mL are clinically significant and typically correlate with reduced sperm production. Below 6 mL indicates severe testicular failure.
Yes, if the cause is hormonal (Hypogonadotropic Hypogonadism). Gonadotropin therapy can significantly increase testicular volume as the seminiferous tubules begin spermatogenesis. However, if the cause is primary testicular failure or genetic, the size may not increase substantially, but sperm retrieval via Micro-TESE may still be possible.
Yes. A large, untreated varicocele creates a state of chronic heat stress and oxidative damage that can cause progressive ipsilateral (same-side) testicular atrophy. Early varicocelectomy can prevent further damage and may partially restore volume.
Low testicular volume demands a systematic, cause-driven evaluation. Dr. Adnan Jabbar's clinic in Lahore provides the most comprehensive male fertility workup in Pakistan.